Presentation description

Antiarrhythmic drugs increase the action potential duration and/or the refractory period by preventing the abnormal electrical activity of atrial fibrillation from propagating further. Although the mechanism of each of these drugs is known, it is still unclear why some of the drugs work better for certain patients than others. The goal of this project is to run patient specific simulations of these drugs to understand what makes certain patients respond better to certain drugs than other.