Presentation description

Alzheimer's Disease (AD) is one of the most common causes of dementia, yet there are limited options for treatment. Microtubule-stabilizing drugs may have the potential for treating AD and altering its pathology by maintaining cytoskeleton structure and stability and preserving functions of neurons, such as axonal transport. This research aims to evaluate changes in hallmark pathophysiological features of AD, namely amyloid plaques and neurofibrillary tangles, after treatment with PTX-conjugate.