Presentation description

In my project, I investigate the impact of mitochondrial damage and dysfunction on cellular energy production, focusing on the crucial role of mitochondrial respiratory pathways in ATP synthesis. Mitochondria, the powerhouses of cells, play a vital role in maintaining cellular function, and disruptions in their activity can lead to reduced ATP production. To address this, our study hypothesizes that intravenous treatment with GJA1-20k can support mitochondrial function in pigs and mice following hemorrhagic shock. This peptide shows potential in protecting mitochondria, reducing oxidative stress, and preventing apoptosis, which are critical factors in improving outcomes during critical conditions.